A role for the FKBP52 protein in treating Alzheimer's disease
New research in humans published this week reveals that the so-called FKBP52 protein may prevent the Tau protein from turning pathogenic. This may prove significant for the development of new Alzheimer's drugs and for detecting the disease before the onset of clinical symptoms.
A study published on-line in the Journal of Alzheimer's Disease, for the first time demonstrates that the FKBP52 protein, discovered by Etienne Baulieu 20 years ago, may prevent hyperphosphorylation of Tau protein, which has been shown to characterise a number of cerebral neurodegenerative diseases, including Alzheimer's Disease (AD).
This work has been carried out by Prof Baulieu and his research team at Inserm (National Institute for medical research in France) with the support of philanthropists who help the Institut Baulieu, based in France.
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